1. Thucydides. The Peloponnesian War. Crawley R, trans. New York,
N Y: Random House; 1951.
2. Margotta R. An Illustrated History of Medicine. Middlesex, England:
Hamlyn; 1967: 86.
3. Traber DL, Herndon DN, Enkhbaatar P, Maybauer MO, Maybauer
DM. The pathophysiology of inhalation injury. In: Herndon
DN, ed. Total Burn Care, Fourth Edition. Philadelphia, PA: W. B.
4. Baile EM, Dahlby RW, Wiggs BR, Paré PD. Role of tracheal and
bronchial circulation in respiratory heat exchange. J Appl Physiol.
1985; 58( 1):212-222.
5. Linares HA, Herndon DN, Traber DL. Sequence of morphologic
events in experimental smoke inhalation. J Burn Care Rehab.
1989; 10( 1): 27-37.
6. Cox RA, Burke AS, Soejima K, et al. Airway obstruction in sheep
with burn and smoke inhalation injuries. Am J Respir Cell Mol Biol.
2003; 29( 3):295-302.
7. Soejima K, Schmalstieg FC, Sakurai H, Traber LD, Traber
DL. Pathophysiological analysis of combined burn and smoke
inhalation injuries in sheep. Am J Physiol Lung Cell Mol Physiol.
8. West JB. Pulmonary Pathophysiology: The Essentials. 6th ed.
Baltimore, MD: Lippincott, Williams & Wilkins; 2003.
9. Smith RP. Chapter 14. Toxic responses of the blood. In Doull J,
Klaassen CD, Amdur MO, eds. Casarett and Doull’s Toxicology, The
Basic Science of Poisons. 1st ed. New York, N Y: Macmillan Company;
10. Navar PD, Saffle JR, Warden GD. Effect of inhalation injury on
fluid resuscitation requirements after thermal injury. Am J Surg.
11. Herndon DN, Traber DL, Traber LD. The effect of resuscitation on
inhalation injury. Surgery. 1986;100( 2):248-251.
12. Piatkowski A, Ulrich D, Grieb G, Pallua N. A new tool for the
early diagnosis of carbon monoxide intoxication. Inhal Toxicol.
2009; 21( 13):1144-1147.
13. Osler T, Glance LG, Hosmer DW. Simplified estimates of the
probability of death after burn injuries: Extending and updating
the Baux score. J Trauma. 2010; 68( 3):690-697.
14. Mlcak R, Desai MH, Robinson E, Nichols R, Herndon DN. Lung
function following thermal injury in children—an 8-year follow
up. Burns. 1998; 24( 3):213-216.
smoke and fumes from conflagration in other
and unspecified building or structure). A total
of 1,258 records were found. Of these records,
988 contained a discharge status, including
668 patients discharged to home, 148 to acute
care/rehab, and 78 sent to skilled nursing
facilities; 95 died. These patients were 56. 6
percent male, on average 45. 9 years of age,
had an average hospital length of stay of 9. 8
days, an average intensive care unit length of
stay of 10. 2 days, an average injury severity
score of 9. 6, and were on the ventilator for
an average of 8. 5 days. Of those patients
tested for alcohol (625), more than half ( 52
percent) tested positive (see figure, page 71).
Activities that generate smoke, such as
a campfire, barbecues, or an old-fashioned
fish boil, may be tantalizing to watch.
However, being confined in an enclosed
space with all smoke and no fire can lead to
a devastating and potentially fatal injury.
Throughout the year, we will be
highlighting these data through brief reports
that will be found monthly in the Bulletin.
The NTDB Annual Report 2014 is available on
the ACS website as a PDF file at www.facs.
org/quality-programs/trauma/ntdb. In addition,
information is available on our website about
how to obtain NTDB data for more detailed
study. If you are interested in submitting
your trauma center’s data, contact Melanie L.
Neal, Manager, NTDB, at firstname.lastname@example.org.
Statistical support for this article has been provided
by Chrystal Caden-Price, Data Analyst, NTDB.
Activities that generate smoke, such as a campfire, barbecues, or
an old-fashioned fish boil, may be tantalizing to watch. However,
being confined in an enclosed space with all smoke and no fire
can lead to a devastating and potentially fatal injury.